Thanks For Stopping By - Friends of Parkinson`s

Transcription

Thanks For Stopping By - Friends of Parkinson`s
P
The
October 2015
Friends of Parkinson’s
Volume 2 Issue 10
arkinson Post
Living Well with Parkinson’s ~ An Integrative Approach
Parkinson's
Alpha-Synuclein Explained
Thanks For
Stopping By
Across The Pond
"All Shook Up"
A Caregiver's
Christmas
Low-Level
Pesticides Linked to
Parkinson's Disease
Friends of Parkinson’s • October 2015 • Page 1
The Parkinson Post
Message From
FRIENDS OF PARKINSON’S
Inside this issue
Subscribe and/or Donate Today (page 8)
Thanks For Stopping By
Hi there. Thanks for stopping by. I’m here to tell you a story about how I’ve
overcome the obstacles that Parkinson’s disease presents daily. It’s a tale of
loss and love, a story of strength and commitment in the face of what simply
is an “incurable progressive neurological disorder.”
Let that reality sink in
for a second before you continue, please.
See, Parkinson’s disease is different for everyone. I presented with “central
pain” stiffness and rigidity while others get the shakes, or tremors. Over
the last four years of having active PD., I’ve found that if I exercise on a
regular basis and eat a proper diet that it is possible to greatly improve the
quality of my life and of those around me. One of the greatest gifts I can
give myself and those on this journey with me are to embrace life. To not let
Parkinson’s disease define me but to let it be the fuel that helps me power
through each day with the hope and dream that someday we will find a cure
for PD and all disease that robs us of our ability to live life to the fullest.
Parkinson’s disease cost me everything, my career, my family and home;
however, it gave me the passion and insight to live the life I was meant to
and to be strong enough to send this message. “Be brave, be bold and thrive
in the life you have.” My Power, through commitment, goes out to my son
and my Katelyn. I may have Parkinson’s disease and it doesn’t have me.
Publisher
Friends of Parkinson’s
Staff Writer
Lisa Vanderburg
Executive Director
Jamillah Ali-Rahman
Contributing Writers
Benjamin Prewitt
D. Joseph Mercola
Advertising
725-222-8181
Low-Level Pesticides Linked
To Parkinson's Disease
By Dr. Joseph Mercola
Page 3
Parkinson's…
Alpha-Synuclein Explained
By Lisa Vanderburg
Pages 4 & 5
across the pond
with Lisa Vanderburg
Page 7
Benjamin Prewitt is an established artist. Diagnosed with Parkinson’s November 2010 at age 40, his approach is gentle, retrospective and always
filled with beauty. A part of Benjamin Prewitt’s artist’s statement tells us
much about the artist when he states, “Art”
reminds us the seemingly
opposite forces of joy and sorrow work in symmetry to inspire us to find
balance in who we are. My greatest sorrow, Parkinson’s disease, does not
define me; I am simply an artist with Parkinson’s. The two are not mutually
inclusive, they just are. Benjamin’s blog and art “A life in progress”
Post
in responds to the Power through Parkinson’s blog soon to be syndicated
and published through the BGF Foundation. www.BenjaminPrewitt.com.
Distribution Manager
Jon Carl Olson
Page 2
Events & Resources
Benjamin
2015
Int’l Research Specialist
Lisa Vanderburg
By Benjamin Prewitt
Page 6
Much love and light.
Editor
Michelene K. Bell
Message From Friends of Parkinson's
Thanks For Stopping By
Featured story
Alpha-Synuclein Explained
By Lisa Vanderburg
Advisory Board
Donna Jensen
Dir. of Operations
Frank Fuentes
Dir. Marketing & Pub. Relations
Lanie Spradlin
Dir. Community Affairs
Office
The Parkinson Post
2400 N. Tenaya Way
Las Vegas, NV 89128
Ph: 725-222-8181
Fx: 702-838-2640
Jim DiFiore
Dir. Business Development
E-mail
Info.ParkinsonPost@gmail.com
Charles Jensen
Legal Advisor
Website
FriendsofParkinsons.org
Shirley Hofmeister
Dir. Emeritus
Tina Drago
Dir. Medical Services
Page 2 • October 2015 • Friends of Parkinson’s
Alpha-Synuclein is a sticky protein found
throughout the body, but the mother lode is in the
brain, where, if all goes smoothly, it's part of a
process that leads to the release of dopamine…
Don’t forget the Sock Hop!
Save this Date: October 24, 2015
The Parkinson Post
Low-Level Pesticides
Linked to Parkinson's
Dr. Joseph Mercola
The risk of Parkinson’s disease
clearly increases with exposure to
certain environmental toxins, such as
“Epidemiological research has
identified several factors that may be
linked to Parkinson’s, including rural
living, well water, manganese and
pesticides."
pesticides. These pesticides, herbicides, and fungicides are potent toxicants that
may cause disruptions or damage the neurological system, including your brain.
What is perhaps most concerning is ambient exposure to pesticides has been found
to increase the risk of Parkinson’s considerably; which was further confirmed by
new research linking the disease to extremely low-level pesticide exposure.
Eleven Commonly Used Pesticides Linked to
Parkinson’s, Even at Low Levels
Last year, UCLA researchers linked a fungicide known as benomyl to PD
[Parkinson’s disease]. The US Environmental Protection Agency (EPA)
banned the chemical after it was determined to be a potential carcinogen.
That study found that benomyl blocks the aldehyde dehydrogenase (ALDH)
enzyme. When working properly, ALDH changes aldehydes, which are
toxic, to dopamine cells, into less toxic compounds. When ALDH is blocked,
however, this transformation does not occur, contributing to the development
of Parkinson’s.
For the most recent study, researchers revealed eleven pesticides in all that
inhibit ALDH and increase the risk of Parkinson’s disease. The effects were
seen even with very low-level exposure – levels lower than are currently being
used in everyday applications. The study’s lead author said:
“We were very surprised that so many pesticides inhibited ALDH and at quite
low concentrations that were way below what was needed for the pesticides to
do their job…
These pesticides are ubiquitous, and can be found on our food supply and are
used in parks and golf courses and in pest control inside buildings and homes.
This significantly broadens the number of people at risk.”
Common Genetic Variant May Raise Parkinson’s Risk
Six-Fold
Certain people appear to be especially sensitive to the effects ALDH-blocking
effects of pesticides. The study revealed people with a common genetic
variant of the ALDH2 gene had a two to six times greater risk of developing
Parkinson’s disease when exposed to pesticides compared to those without the
genetic variant. The study reported:
“ALDH inhibition appears to be an important mechanism by which these
environmental toxins contribute to Parkinson’s pathogenesis, especially in
genetically vulnerable individuals.”
Unfortunately, unless you’ve been specifically tested, you’ll have no way of
knowing whether you have this particular genetic variant. Moreover, even
those who do have it will have a hard time completely avoiding pesticide
exposure, since their use is ubiquitous.
Can We Still Say Parkinson’s Disease Has No
Identifiable Cause?
Parkinson’s disease is still classified as idiopathic, meaning it has no
identifiable cause. However, one reason it is likely on the rise is due to many
environmental toxins that now bombard your body on a daily basis, with
pesticide exposure becoming an undeniable risk factor. According to the
Parkinson’s Disease Foundation:
“Epidemiological research has identified several factors that may be linked
to Parkinson’s, including rural living, well water, manganese and pesticides.
Some studies have demonstrated that prolonged occupational exposure to
certain chemicals is associated with an elevated risk of PD. These include
the insecticides permethrin and beta-hexachlorocyclohexane (beta-HCH), the
herbicides paraquat and 2,4-dichlorophenoxyacetic acids and the fungicide
maneb. In 2009, the US Department of Veterans Affairs added Parkinson’s to a
list of diseases possibly associated with exposure to Agent Orange.”
Separate research has further revealed ambient exposure to organophosphate
pesticides also increased the risk of developing Parkinson’s. Rotenone and
paraquat are two additional pesticides linked to an increased risk of Parkinson’s
disease, and both are lipophilic, meaning they resist breaking down in water
and accumulate in your fat. Both are known to cross your blood-brain barrier.
Dousing our crops with large amounts of glyphosate, the active ingredient in
Monsanto’s broad-spectrum herbicide Roundup, may be another factor in the
rising rates of Parkinson’s disease. These residues are found in virtually all
food containing genetically modified ingredients.
Avoiding pesticide exposure around your home, in your community, and via
the food you eat, is clearly important for reducing your Parkinson’s risk, as
is reducing your exposure to environmental toxins. Exposure to industrial
solvents, including TCE, a common degreasing agent and dry-cleaning
chemical, has also been linked to Parkinson’s disease for instance. Therefore,
along with avoiding the use of solvents and pesticides in your home and garden,
eating organic foods as much as possible will help you avoid unnecessary
exposure to these chemicals.
Another important and often-overlooked environmental risk factor is “silver”
amalgam dental fillings, which actually are more than half mercury. Mercury
becomes a biochemical train wreck in your body, causing your cell membranes
to leak, and inhibits key enzymes your body needs for energy production
and removal of toxins. Mercury toxicity can lead to major inflammation and
chronic illnesses such as PD.
Healthful Lifestyle May Lower Parkinson’s Risk
Parkinson’s is a neurological disorder in which neurons in a region of dopaminecontinued on page 6
Friends of Parkinson’s • October 2015 • Page 3
The Parkinson Post
Parkinson’s
by Lisa Vanderburg
Alpha-Synuclein Explained
K
eep it simple, stupid!’...that’s my motto. Don’t get me
wrong – not for you, but for ME, because it takes ages to
understand, much less absorb or translate! Being two stories
short of a high-rise presents serious challenges and lots of ‘duh’
moments, but I think I got it?
is aggregation - crap everywhere that gum-up the works. These
aggregates are commonly known as Lewy bodies. Lewy bodies are
present in a variant of Alzheimer's disease also known as Lewy
body dementia, another disease known as multiple system atrophy
or ‘neurodegenerative’, and, of course, Parkinson’s disease.
So for all you peeps out there that wanna know the hoo-ha about
alpha-synuclein…here it is.
Now, that’s the simple version….without reference to other
players like Tau and A-Beta proteins, or DNA strands,
mitochondria, enzymes, or the many genes already identified as
biomarkers for the disease. This will have to do to discuss the
major issue, which is:
Let’s gets a couple of abbreviations out of the way first:
a-SYN = Alpha-Synucein
PD = Parkinson’s
‘Aggregates’ are basically debris or detritus; organic matter produced by the decomposition of organisms.
Alpha-Synuclein is a sticky protein found throughout the body,
but the mother lode is in the brain, where, if it all goes smoothly,
it’s part of a process that leads to the release of dopamine – that
wonderful stuff that makes us feel good, controls movement
and autonomic systems, all with the agility of a synchronized
swimmer.
Ideally, the a-SYN protein appears to function in the loading of
vesicles (see graphic below) - small spherical bits of membrane
that carry neurotransmitter molecules - to the pre-synaptic end of
a neuron, which bumps
Presynaptic
into the business end
Membrane
of another neuron,
synaptic
allowing for the neurovesicle
transmitters in the
synaptic cleft
vesicles to be released
neurotransmitter
into the synapse.
Postsynaptic
Membrane
The top is the presynaptic neuron; the
bottom, postsynaptic.
They are both separate neurons in this chain gang.
Okay. So what’s the big dealeo? Glad you asked.
In Parkinson’s and specific other neurodegenerative diseases, it’s
been found that a-SYN proteins are ‘misfolding’, creating clumps
or aggregates. Like almost all proteins, after a-SYN is synthesized
by the cellular machinery (i.e. ribosomes – they build long chains
of amino acids, one at a time – exhausting!), it is supposed to
fold in a certain way; the entire function of a-SYN is dependent
on proper folding. So, if the folding goes wrong, then the protein
fails to function – simple! The fall-out of this wayward behaviour
Page 4 • October 2015 • Friends of Parkinson’s
So it’s rather like loading a musket rifle or cannon; exact amount
of powder shove a lead ball down the barrel specific grade of
powder into the pan before closing the frizzen = success!
Do it wrong = ka-boom…
or maybe no head…
Nah…bit sorta, er…permanent?
Worse, these deviant, delinquent aggregates are not the only
bits of junk that scupper any healthy a-SYN from assisting with
neurotransmitter release from neurons. There’s the possibility
of too much a-syn being produced as well, and that excess of
a-SYN would lead to toxicity. Either way, its accumulation is
a big problem in 90–95% of Parkinson’s patients. The other
5% are those that have direct-inherited disease (genetically
passed down from parent to child) and for them, a-SYN is not
considered a big player.
Oh…did I mention, this toxicity is specific to neurons that
release the neurotransmitter dopamine, and the aggregation
may even be protective in other types of neurons? Okay,
that makes no sense. I think what I’m saying here is that, on
a cellular and molecular level, waste product is normal and
dealt with in an orderly fashion. Not so with these a-SYN,
simply because there’s too much junk. And another thing we
have to throw into the mix; oxygenation.
Say hello to ‘free-radicals’, which is what the dopamine
neuron-specific toxicity appears to involve: the formation of
The Parkinson Post
Alpha-Synuclein is a sticky protein found
throughout the body, but the mother lode is in the
brain, where, if it all goes smoothly, it’s part of a
process that leads to the release of dopamine…
reactive oxygen, which can evolve during the breakdown of
excess dopamine that builds up when it cannot be released
from the neuron.
Pharmaceutical companies like Neuropore Therapies, are teaming up with Belgian drug maker UCB, and QR Pharma, Inc. has
something in the pipeline too.
Okay, okay, but at a cellular level, it’s poison. It’s caustic and can cause the iron in
our cells to basically go rusty. Yuck.
Here’s where I may have to kneel on grits (ol’ Southern
hospitality!):
A similarly unexpected finding; one
that’s got everybody hot and bothered,
generating a great deal of interest, is
that misfolded a-SYN are also found in
our gut and olfactory (that’s ‘nose’ to
you and me) system, which means they
travel. To show you the significance of that, Patrik Brundin, MD,
in 1988 received his PhD on intracerebral transplantation in PD.
By 1992, he was part of a team that started neural transplants in
PD patients. That worked well…for a time. But, and here’s the
important bit, in 2008, they found Lewy bodies (these misfolded
a-synuclein clusters) in the significantly younger (chronologically)
grafts. Well, that opened up a whole NEW can o’ worms. Could it
be this was Prion disease – neurodegenerative disorders causing
inflammation and infection? Or maybe sub-structural faulty
amino-acids could be a factor in a-SYN clumping? The body
naturally deals with waste products – even on a cellular level, but
these aggregates are too numerous.
More importantly, it meant that, at some point during their travels, they were passing on a mutation to their buddies….and in
turn suggested at some stage in this process, they were vulnerable. It was then a completely reasonable idea to think that bacteria might be involved. What kills bacteria? Antibodies, for one.
That has led to some remarkable potential offerings in the
Pharmaceutical field like NeuroPhage Technologies’ drug
candidate NPT001 (bacteriophage M13), which are in the process
of filing with the FDA; the first human trials expected in 2016.
But, it requires administration to the brain directly, as crossing
the blood-brain barrier (BBB) is a SOB. Most antibodies are too
large to get in.
Another offering is ICBI Technologies, and their SMART molecule
(Specific Molecular Architecture for Recognition and Therapy Molecule), which has proven demonstration of BBB uptake in mice.
Parkinson’s Resource Organization has been working closely with
this Pharma since 2012. They keep us posted by way of ‘the Road
to the Cure’ http://www.parkinsonsresource.org/featured-posts/
august-2015-update-on-the-road-to-the-cure/. That will be the
latest, but they keep a running update from back to 2012.
All the above have, at some stage or another, had significant
grants from people like the MJFox Foundation.
And I shoved poor MJ’s mug on three
of my posts. Why? Because I wanted
all the Orgs to work together under
the umbrella of, say the World Parkinson’s Coalition. Interestingly, it
was Jo Rosen, Founder & President
of Parkinson’s Resource Organization, CA who schooled me on why all
the PD groups, Assocs., Foundations
& Institutes do NOT work well together…has a lot to do with agendas. Bleedin’ politicking!! Apologies, Michael J Fox! What did you
ever do to attract my beady eye? Well…you got Parkinson’s for
a start, and then went on to establish the biggest PD non-profit
globally. And earnestly, those posts were for awareness, not target-practise. I think MJ himself would see that!
But Jo Rosen – one feisty and savvy warrior told me “If ICBI had
$5M today, it could be at human clinical trials in 18 to 24 months.
I’ve already spent 3.5 years on this, Lisa. Imagine, had people
opened their minds and pocket books 3.5 years ago, we would
have been where we are still looking to be...’’ Now $5 million is a
drop in the ocean in the scheme of things; chump-change for some.
Anyway, after 30 years of basically the same old drug – Levadopa,
somebody takin’ out the trash might well save us!
With sincere thanks to Dr. Anuj Mankad, Molecular and Medical
Geneticist, who so patiently picked up after I’d thrown all my
toys outta the pram – thanks Doc, for the schooling!
Lisa describes herself as a ‘chaotic and vaguely
confused pit-bull’ who would ‘cheerfully
throttle Parkinson’s Disease’ if she could! She
is a European writer and the carer [caregiver]
for her husband Pete, who she sees as a
‘shameless, wanton optimist’ – she knows what
a blessing that is! She has now joined us as the
International Research Manager and Staff
Writer for Friends of Parkinson’s.
Friends of Parkinson’s • October 2015 • Page 5
The Parkinson Post
Lisa Vanderburg
International Research Specialist for Friends of Parkinson's
Across Pond
the
Sublime and Redonkulous: Parkinson's
Well hello, fellow oddballs. I say that because PwP (people with
Parkinson’s) and PwP/SD (that’ll be their caregivers or ‘Slave-Drivers’)
are all oddballs. It’s not the ‘downside’ of Parkinson’s but quite the
reverse! No matter if, we are the detained or detainee, it’s Parkinson’s
that rules the roost over both the afflicted and affected. It’s baffling,
befuddling, ungainly and just plain confounding. So demanding is
Parkinson’s, that it so often leaves us depleted of any semblance of
control, there’s not much else to do but LAUGH! Laugh so hard, we can
control out tears (we call it schlapp-lakking– great word!).
My sister and me would so often be overcome by schlapp-lakking always in the most exhausting circumstances. I’m one and a half years
her junior, and was helping her move from her house (she’d raised her
kids in…and she was a HOARDER and a half!), in a hurry. I’m a stocky
pit-bull. She’s a stick insect with a seriously over-blown sense of her
physical acumen.
CAVEAT - this is just my humour; I even dug a hole for her cat (very
much alive then). It sounds wrong, but the British are so obsessively
compulsive about animals. Vetting potential humans for re-homing
involves criminal background, sodium pentothal, lie detector tests, and
signing your life away; because those in control will visit said adoptee
and give it stuffed, fluffy rodents to play with while under ‘observation’.
However, this mog was running out of time – no takers, and she was
moving to a flat (apartment) which does not take animals - few do, and
you have to make a pact with Beelzebub and sign your kidneys away.
Thus, it was reasonable to assume, at the 11th hour, Fluffy was not going
to make it. He did…just, and I had to fill the bloody hole in.
I digress, sorry! Last couple of days, and with the meter ticking on
exchange, she wanted to take a ‘couple’ of wee plants. I groaned noisily,
but can never refuse her anything. So we end up schlepping several
TREES in tubs probably weighing in at 300+lb. per – UP the steep
garden to the waiting car…that’s when schlepping sets in – fits of sidebusting giggles turn into uncontrollable roaring – followed by a quick
bathroom break. Ladies who have born children will understand that
heavy lifting whilst laughing hysterically will lead to ‘accidents’! A job
that would’ve taken a couple of blokes 10 minutes, took us half a day,
but worth every moment!
continued from page 3
Low-Level Pesticides…
Dr. Joseph Mercola
producing cells within your brain, known as the substantia nigra, required for
normal movement, begin to die. As the disease progresses, symptoms include
increasingly slow motor function, tremors when at rest, muscle rigidity, and
abnormalities in your walk. Up to one million Americans currently have
Parkinson’s and 60,000 more are diagnosed each year.
As a disease that currently cannot be cured, prevention of Parkinson’s is crucial.
People over 60 years of age used to make up the majority of those afflicted, and
with increasing lifestyle factor risks like chronic exposure to pesticides and a
generally increasing toxic burden, cases are now being reported regularly in
people under 50 and even under 40 years of age.
Parkinson’s is strongly related to lifestyle choices, which is good news because
it means there are steps you can take to modify your risk. Eating a varied
whole-foods diet that includes healthful veggies like peppers appears to be one
simple way to lower your risk, especially since a lack of folate is also linked to
Parkinson’s (and veggies are the only source of this important vitamin; most
vitamins contain the semi-synthetic analog known as folic acid). Additionally,
the following nutrients are also important and may help you to significantly
lower your risk:
Vitamin D: There is a correlation between insufficient levels of vitamin D
and the development of early Parkinson’s disease. It used to be thought that
vitamin D deficiency is a symptom of Parkinson’s, but recent research squarely
implicated vitamin D deficiency as one of the causes of Parkinson’s. The
best way to optimize your vitamin D level is through appropriate midday sun
exposure or a safe tanning bed, as optimizing your body’s own production
virtually eliminates any risk of overdose.
Animal-based omega-3 fats: These may protect against Parkinson’s by
preventing the misfolding of a protein associated with a gene mutation in
neurodegenerative diseases like Parkinson’s and Huntington’s. Animalbased omega-3 fats contain two fatty acids crucial to human health, DHA and
EPA. Most of the neurological benefits of omega-3 oils are derived from the
DHA, which is one of the major building blocks of your brain. Adding omega-3
fats to your diet, via wild-caught fish or a supplement like krill oil, is important.
Coenzyme Q10 (CoQ10): One study found that the progression of Parkinson’s
disease was significantly slower in people taking the highest dose of CoQ10. If
you’re taking statin cholesterol-lowering drugs, this issue is particularly important
as these medications deplete your body of CoQ10, making supplementation with
CoQ10 (or ideally, the reduced form, called ubiquinol) important.
What was I saying? Ah, yes…something about Parkinson’s! Over the coming
months, I intend to offer you my ‘take’ on living with PD. That’s because I
have a 6’3’’ 230lb festinating bloke who I happen to be the caregiver for. He’s
Shaky Pete - Puff Daddy – Butterball – my husband Pete who is in his 16th
year of PD.
Green Tea: Green tea’s polyphenols offer neuroprotection that might benefit
Parkinson’s patients. Caffeine, which is doparminergic (stimulates dopamine
release), may be one more reason why green tea has also been linked to a lower
Parkinson’s risk. In fact, the daily caffeine equivalent in two to four cups of
coffee may modestly improve Parkinson’s movement symptoms as well.
vvvvv
Finally, your best bet for minimizing health risks from pesticide exposure is
to avoid them by eating organic as much as possible and investing in a good
water filtration system for your home or apartment. If you know you have been
exposed to pesticides, the lactic acid bacteria formed during the fermentation
of kimchi may also help your body break down pesticides. By including
fermented foods, like kimchi, in your diet, is a wise strategy to help detox the
pesticides that enter your body.
Lisa Vanderburg is the European International
Research Specialist and Staff Writer for Friends of
Parkinson’s.
Wishing You Health and Happiness
From Across The Pond!
Lisa Venderburg
Page 6 • October 2015 • Friends of Parkinson’s
vvvvv
Dr. Mercola authored two New York Times best sellers, The No-Grain Diet and
The Great Bird Flu Hoax. An osteopathic physician (DO), his primary passion
is transforming the conventional medical paradigm in the United States. Visit
www.DrMercola.com, Mercola.com.
Events & Resources
October
TAI CHI FOR BALANCE, Mondays & Wednesdays,
11am. West Flamingo Active Adult Cntr. 6255 W,
Flamingo. 702-455-7742.
MOVE I EXERCISE CLASSES, Tuesdays &
Thursdays, 11:15am to 12:15pm, (seated exercise
for anyone with neurodengerative disorders) Lou
Ruvo Center for Brain Health. 888 W. Bonneville,
call Lakita Arrington. 702-483-6032.
MOVE II EXERCISE CLASSES, Tuesdays &
Thursdays, 12:30pm to 1:30pm, (standing
exercise for anyone with neurodengerative
disorders) Lou Ruvo Center for Brain Health.
888 W. Bonneville, contact Lakita Arrington.
702-483-6032. ($10 drop in price).
ZUMBA GOLD, Wednesdays, 9-10am. 2651 Paseo
Verde Pkwy, Suite 180. Contact Kim Riddle, 702616-4902. ($5/per session or 5 sessions for $20).
AGELESS WOMAN WORKOUT, Tuesdays &
Thursdays, 9-10am. 2651 Paseo Verde Pkwy,
Suite 180. Contact Kim Riddle, 702-616-4902.
($5/per session or 5 sessions for $20).
GENTLE YOGA, Tuesdays & Thursdays, 10-11am.
2651 Paseo Verde Pkwy, Suite 180. Contact
Kim Riddle, 702-616-4902. ($5/per session or 5
sessions for $20).
AEROBIC FUN EXERCISE CLASSES, Mondays,
Wednesdays & Fridays, 9:30am. Fern Adair
Conservatory of the Arts, 3265 E. Patrick Lane.
TAI CHI FOR BALANCE, Fridays, 9-10am.
Desert Breeze Community Center, 8275 Spring
Mountain Road, LV. 702-455-8334. Facilitator
Tamalyn Taylor MS.
Dance for Parkinson's, Wednesdays, 12:301:30pm, Las Ventanas (open to the public).
YOUNG ONSET PD SUPPORT GROUP of Southern
Nevada, Cidney Donahoo, 702-326-6831.
CidneyDonahoo@cox.net.
DBS SUPPORT GROUP of Southern Nevada, Kip
Smith, 702-755-5704. kslvnv@gmail.com.
LET'S MEET & EAT FOR CAREGIVERS,
• 2nd Friday, 10am, H2U Mountain View Office,
3150 N. Tenaya Way, Suite 114. Call 702-3814141 for info.
• 3rd Tuesday, 2pm,Vida Training Center, 1785 E.
Sahara Ave., Suite 430. Call 702-381-4141.
Veteran'S INVOLVED PARKINSON'S (VIP), 1pm
(1st Tuesday) Friends of Parkinson's Office,
2400 N. Tenaya Way. 702-381-4141.
PWP BRUNCH BUNCH,
• 1st Saturday, 9am. Mountain View Hospital,
3100 N. Tenaya Way, First floor. 702-381-4141.
PWP BRUNCH BUNCH,
4th Tuesday, 1:30pm. H2U Mountain View
Office, 3150 North Tenaya Way, Suite 114. 702381-4141.
HEALTHSOUTH DESERT CANYON HOSPITAL,
The Parkinson’s Disease Rehabilitation Program provides a full range of rehabilitative
services aimed at meeting the global range of
medical, psychological and functional needs of
Parkinson’s patients. 9175 W. Oquendo Rd., Las
Vegas. For additional information please call
702-252-7342.
SPECIAL ANNOUNCEMENT
AbbVie Usa Llc
Speak at each Friends of
Parkinson's Support Groups
call 702-381-4141
to
PPPPPPP
Saturday March 26, 2016
FUNNY BUNNY RACE ONLINE
Early sign ups @
www.Active.com
Make this day special and then
pay-it-forward!
Location: The Elks Lodge, 4100
West Charleston Blvd, LV
Friends of Parkinson’s • October 2015 • Page 7
Page 8 • October 2015 • Friends of Parkinson’s